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Septic cardiomyopathy (SIC) In a model of LPS-mediated septic cardiomyopathy, some researchers revealed that LPS caused ferroptosis and septic cardiac injury by downregulating GPX4 protein levels and increasing COX-2 [93]
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These factors, including vWF (von Willebrand factor), ADAMTS13, aPTT, FVIII, FXI, FVII, FX, ETP, PAI-1, protein C, and plasmin, were grouped by their roles in platelet adhesion, intrinsic and extrinsic pathways, common pathways, and fibrin dissociation
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